Research Updates

posted on October 3, 2013 - 9:15am
In its Summer 2013 round of grants, MDA awarded 31 new grants totaling $8.5 million. 
posted on October 3, 2013 - 9:01am
Quest Vol. 20, No. 4
Note: To receive a monthly email summary of MDA research news, be sure to sign up for MDA News. ALS (amyotrophic lateral sclerosis)
posted on July 1, 2013 - 9:04am
Quest Vol. 20, No. 3
Note: To receive a monthly email summary of MDA research news, sign up for MDA News. General news
posted on April 1, 2013 - 9:03am
Quest Vol. 20, No. 2
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A follistatin gene therapy compound gains orphan drug status; potential improvements are reported for utrophin-based therapies, exon skipping and stem cell transplantation

posted on December 27, 2012 - 5:00am
Update (Jan. 23, 2013): The "Building better utrophin" section was updated to reflect the availability of a Jan. 22, 2013, press release from the University of Missouri. Below is a wrap-up of recent research news about the development of therapies for Duchenne, Becker and limb-girdle muscular dystrophies.

The drug improved heart function and survival in mice with lamin A/C mutations, which cause lamin A/C-related Emery-Dreifuss MD and type 1B limb-girdle MD

posted on May 18, 2012 - 2:00pm
Update (June 1, 2012): This story was updated to include the availability of a podcast on this topic.

Top researchers are discussing notable developments in neuroscience, including research in many neuromuscular diseases, at the annual American Academy of Neurology meeting April 21-28

posted on April 24, 2012 - 4:00am
Top scientists and clinicians from around the world are discussing the latest research in neuroscience, and the care of individuals with nerve and muscle diseases, at the 2012 annual meeting of the American Academy of Neurology in New Orleans, April 21-28.

The shorter-than-normal dystrophin protein produced via an experimental exon-skipping drug allows proper localization of proteins at the muscle-fiber membrane

posted on February 8, 2012 - 2:45pm
Shortened versions of the muscle protein dystrophin — produced by skipping a section of genetic instructions called exon 51 — appear to be functional, says a new report from the United Kingdom.